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Potentially major shifts in privacy norms are taking place as a result of advances in genetic technologies. This chapter identifies a spillover effect in the form of the inadvertent emergence of new norms and introduces an original typology developed in response to these new norms regarding privacy. It focuses on the emerging practice of compelling access to genetic information of biologically related persons to gain information about a particular individual. This chapter highlights the recent practice in child lead paint poisoning cases in which defendants seek to discover medical and I.Q. records of biologically related non-parties to establish alternate genetic causation of low I.Q. It concludes that greater attention should be given to the spillover effect and the emergence of shadow norms.

Since the beginning of the 20th century environmental health researchers have known about the association between toxicant exposure and disease. However, that knoweldge has not been well integrated into mainstream medicine. Shedding light on why is the focus of this chapter.

To shed light on this issue I analyze the 2011 American Academy of Pediatrics’ clinical practice guidelines for Attention Deficit/Hyperactivity Disorder (ADHD), focusing specifically on the omission of environmental health research pertaining to ADHD symptoms and exposures, such as lead and mercury.

I found that while environmental researchers have been documenting the link between lead and ADHD for over forty years, the American Academy of Pediatrics has completely omitted this research from its 2012 clinical practice guidelines. Moreover, I argue this omission can be traced to competitive pressures to protect medical jurisdiction, and a reductionist worldview that emphasizes treatment over prevention.

This is the first attempt to analyze the way clinical practice guidelines help reinforce and perpetuate dominant medical perspectives. Moreover, to shed explanatory light, this chapter offers a synthetic explanation that combines materialist and ideological factors.

Beyond the specific case of ADHD, this chapter has implications for understanding how and why environmental health research is omitted from other materials produced by mainstream medicine, such as materials found in the medical school curriculum, continuing medical education, medical journals, and on the medical association web sites.

This chapter is structured for teaching young learners with other health impairments in special education. Under the Individuals with Disabilities Education Improvement Act (IDEA, 2004), other health impairments represent chronic or acute health problems such as asthma, attention deficit disorder or attention deficit hyperactivity disorder (ADHD), diabetes, epilepsy, a heart condition, hemophilia, lead poisoning, leukemia, nephritis, rheumatic fever, and sickle cell anemia and adversely affect a child’s educational performance. The chapter is organized around definitions, prevalence, etiologies, intervention strategies, and teaching considerations for selected disabling conditions in this disability category.

This chapter chronicles some of the early years of the author growing up in the racially segregated South Alabama and its influence on his thinking about race, environment, social equity, and government responsibility and his journey to becoming an environmental sociologist, scholar, and activist. Using an environmental justice paradigm, he uncovers the underlying assumptions that contribute to and produce unequal protection. The environmental justice paradigm provides a useful framework for examining and explaining the spatial relation between the health of marginalized populations and their built and natural environment, and government response to natural and man-made disasters in African American communities. Clearly, people of color communities have borne a disproportionate burden and have received differential treatment from government in its response to health threats such as childhood lead poisoning, toxic waste and contamination, industrial accidents, hurricanes, floods and related weather-related disasters, and a host of other man-made disasters. The chapter brings to the surface the ethical and political questions of “who gets what, why, and how much” and why some communities get left behind before and after disasters strike.

Research at the intersection of neurotoxicology, cognitive neuroscience, genetics, and behavior – “toxicogenomics” – promises to improve understanding of behavior and public policies. Many toxicants modify neurotransmitters with effects depending on genotype, development, and experience. For example, lead and manganese downregulate dopaminergic or serotonergic function, weaken behavioral inhibition, and are positively correlated with hyperactivity (ADHD), substance abuse, or violent crime. Within this context, the chapter argues that neurotoxicology holds great promise for improving public policies in areas such as criminal justice and education. After discussing research methods, the chapter surveys previous work in neurotoxicology as it pertains to educational outcomes and to crime. Data are summarized linking: (1) children's blood lead levels with poor educational performance; (2) hair and blood levels of heavy metals with violent crime; and (3) uptake of multiple toxicants with pollution from abandoned mines. The chapter concludes by drawing lessons for ongoing research agendas at the intersection of neurotoxins, behavior, and policy.

Although established theoretical models suggest that race differences in physical health are partially explained by exposures to environmental toxins, there is little empirical evidence to support these processes. We build on previous research by formally testing whether black–white differences in self-rated physical health are mediated by the embodiment of environmental toxins.

Using cross-sectional data from the National Health and Nutrition Examination Surveys (2007–2008), we employ ordinary least squares regression to model environmental toxins (from urine specimens) and overall self-rated health as a function of race and ethnicity. We employ the Sobel test of indirect effects to formally assess mediation.

Our results show that non-Hispanic black respondents tend to exhibit higher levels of total toxins, lead, and cadmium in their urine and poorer physical health than non-Hispanic whites, even with adjustments for age, gender, and socioeconomic status (SES). Our mediation analyses suggest that blacks may exhibit poorer physical health than whites because they tend to embody higher levels of cadmium.

Research limitations include cross-sectional data and restricted indicators of SES.

This study contributes to previous work by bridging the fields of social epidemiology and environmental inequality and by formally testing established theoretical models.

The percentage of North Americans who have one or more physical disabilities continues to rise. Specifically, the percentage of people with ambulatory disabilities, cognitive disabilities, and other health impairments is increasing every year. This phenomenon calls for pragmatic measures to help provide better transition and related services to students with physical disabilities and other health impairments. It is anticipated that well-planned collaborative transition services provided to students with physical disabilities and other health impairments will result in improved quality of life and independent living in the community. In this chapter, we discuss transition and transition-related services, supporting legislation for persons with disabilities, transition from rehabilitation centers and hospitals to job settings and community-based programs.