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This study aims to empirically examine how the Khmer Rouge regime (1975–1979) in Cambodia continues to affect the health of the second generation.

The 2000 and 2005 Cambodia Demographic and Health Surveys were used in the analysis. The study sample were women with a child/children in 2000/2005. The sample population was identified according to whether the person was in utero “91 months or earlier before the Khmer Rouge regime,” “46–90 months before the Khmer Rouge regime” and “1–45 months before the Khmer Rouge regime” and during the Khmer Rouge regime. The authors then regressed the size of babies of the targeted population on the timing of the mothers being in utero.

Mothers who were in utero during the regime had a higher likelihood of giving birth to smaller-than-average babies. Additionally, mothers born in the areas that had a higher probability of death of children aged five or under during the regime were at risk of giving birth to smaller-than-average babies if they were in utero during that time.

This is the first paper to assess the impact of the Khmer Rouge regime on the health of the second generation.

The purpose of this review of the literature is to provide data about flaxseed intake during pregnancy and/or lactation and its effects in the offspring from birth to adulthood.

This review includes up-to-date information from evidence-based sources on flaxseed intake and its by-products, during pregnancy and lactation and its effects on male and female offspring, from post-weaning until adulthood. Topics included are effects on body mass; glycaemic metabolism; lipid profile; blood pressure and aortic structure; reproductive system and brain tissue.

The main effects of flaxseed or its by-products were observed in the cardiovascular system, where a lipid profile improvement and minor aortic remodelling were noticed, and in the cerebral development, where greater n-3 PUFA incorporation in the brain was detected.

The research done in this study, to understand the offspring response that were early exposed to the flaxseed components during pregnancy and lactation, may be the first step toward guiding future strategies for recommending the use of this seed during the offspring’s perinatal period.

Prenatal exposure to adverse conditions is known to affect health throughout the life span. It has also been shown that health is unevenly distributed at advanced ages. This chapter investigates whether health inequalities at old age may be partially caused by prenatal circumstances. We use a sample of people aged 71–91 from eight European countries and assess how shocks in GDP that occurred while the respondents were still in utero affect four important dimensions of later-life health: cognition, depression, functional limitations, and grip strength. We find that early-life macro-economic circumstances do not affect health at advanced ages, nor do they affect inequalities in health. In additional analyses, we show that the least healthy people may not enter our sample as the probability of dying before reaching age 71 is high, and mortality rates among those who were prenatally exposed to adverse GDP shocks are higher. We conclude that selective mortality may mask effects of early-life circumstances on health and health inequality at old age.

This study contributes to the understanding of the health effects of pesticides exposure and of how pesticides have been and should be regulated.

This study presents literature reviews for the period 2000–2013 on (i) the health effects of pesticides and on (ii) preference valuation of health risks related to pesticides, as well as a discussion of the role of benefit-cost analysis applied to pesticide regulatory measures.

This study indicates that the health literature has focused on individuals with direct exposure to pesticides, i.e. farmers, while the literature on preference valuation has focused on those with indirect exposure, i.e. consumers. The discussion highlights the need to clarify the rationale for regulating pesticides, the role of risk perceptions in benefit-cost analysis, and the importance of inter-disciplinary research in this area.

This study relates findings of different disciplines (health, economics, public policy) regarding pesticides, and identifies gaps for future research.

There needs to be an increased recognition of fetal alcohol spectrum disorder (FASD) in services that deal with young people with disruptive and offending behavior, not just those services that deal with adolescents with a recognized intellectual disability. The paper aims to discuss these issues.

This is a general review of the current available evidence on FASD and how it is likely to predispose affected young people to have contact with secure mental health services and the criminal justice system.

FASD is likely to have become a more common cause of intellectual disability and behavioral disturbance but the history of significant alcohol exposure in utero if often missed. There is evidence that the hyperactivity is less responsive to psychotropic medication and may represent a different condition to conventional ADHD. However the majority of those affected are in the low normal IQ range.

There is so far very limited research in what is likely to be a relatively common disorder with significant costs to criminal justice, mental healthcare and social services. Epidemiological information from the UK is lacking and urgently needed.

Professionals who work with mentally disordered young people need to be more aware of FASD and its potential contribution to the problems and disabilities in their population.

Social workers, foster carers and adoptive parents need to be more aware of FASD and how it can contribute to the breakdown of social care.

There is currently no other review of FASD and the implications for criminal justice, secure mental health and social care for young people.

Lead is a well-established environmental contaminant that over the last 50 years has become recognized as a neurotoxin with its greatest concern for the developing child (i.e. both in-utero and postnatally). What is problematic is that children exposed to lead often come from lower socioeconomic status (SES), are largely Black communities and are further at increased risk for developing adverse childhood experiences (ACEs). The literature on ACEs had focused much on trauma, single parenting, child abuse, lack of finances and stress, etc., but has not considered the intersectionality of these ACEs as risk factors within environmental neurotoxic exposures such as lead poisoning. This is important as most low SES communities are Black. In particular, within the New York City Housing Authority (NYCHA), Black families have been neglected of proper lead-abatement to their apartments for nearly 70 years.

This is a viewpoint/perspective paper that examines the lived experiences of Black folxs in NYCHA through a Black critical theory (BlackCrit) and antiblackness framework pertaining to ACEs, and lead poisoning within the NYCHA system of New York City. This perspective paper draws upon the last three years of news reports, five decades of publicly available data sets from NYCHA and the comptroller to raise an awareness of how Black children are treated by NYCHA generation after generation which can be argued as a mass atrocity against NYCHA residents. Furthermore, the systematic and institutionalized racism and environmental injustices by NYCHA and the state can also be considered as a crime against humanity. As such, BlackCrit could help to position awareness, advocacy and knowledge about Black folxs residing in NYCHA to achieve fair, safe and affordable public housing to experience Black joy across future generations.

Thus, rather than civic and state government response efforts focusing their full attention and resources to serving and supporting individuals affected by ACEs they should equally consider the environments in which Black people live and also allocate funds proportionally to address these areas often overlooked. Moreover, proportions of these funds should be redirected especially to lead-abatement and removal of known sources of lead exposures, evaluation of suspected sources of lead exposures (i.e. drinking water, baby food and formula, children’s juice and cereal products, superfund and other waste sites, electronic recycling plants, etc.) and accompanied by all affected children undergoing full and comprehensive neuropsychological testing and follow up studies paid for by the state. The goal should have two fundamental objectives: (1) accepting accountability for failing to address these preventable neuropsychological issues directly affecting Black children generation after generation and (2) offering the proper waived or reimbursable supports and resources to help Black children sustain the best quality of life (QOL) trajectory possible when diagnosed with lead poisoning.

The manuscript is a viewpoint/perspective paper grounded in BlackCrit and an antiblackness framework. There are ample public news reports and public data available from NYCHA on these matters over the last three years. However, the scope of this paper was not to delve too deep into these numbers per se, but rather to address the concerns leading up to and arguably contributing to, at least in part, to these numbers of lead-exposed Black children in NYCHA. Lead poisoning has never been considered as an ACE and its relationship to mass atrocity research is novel which may pave a new avenue for research of this kind through the utility of BlackCrit and antiblackness framework to support and advocate for change so that Black children can be provided with a basic human right of safe housing and experience Black joy.

BlackCrit has not been used in the context of lead poisoning research. Mostly individuals and families of middle- and low-income have been studied in the context of poverty and lead poisoning. However, many people who live in poverty, in public housing, within New York are Black. Thus, Black children are generation after generation exposed to unaddressed lead-abatement and it appears that now more than ever BlackCrit should become the framework for how this work should be discussed in the literature to raise awareness to state governments regarding Black folx's persistent lead poisoning, NYCHA's neglect and mass atrocity research as a long overdue advocacy effort to bring the necessary voice, authentic narrative, and actual knowledge of the lived experiences of Black families in NYCHA with lead poisoning.

The goal of this viewpoint/perspective paper should have two fundamental objectives (1) NYCHA and New York State accepting accountability for failing to address these preventable lead poisoning issues directly affecting Black children; and (2) offering the proper support and resources to help Black children sustain the best QOL trajectory possible when diagnosed with lead poisoning.

Lead poisoning research has never been approached through a mass atrocity and BlackCrit framework and perspective. This is the first report on bridging these fields within the context of NYCHA public housing neglect of lead-abatement and continued poisoning of current and future generations of Black children. This failure of NYCHA lead-abatement contributes annually to economic loss in New York State for many years to come which could be entirely avoided.

Purpose – This chapter examines the literature on the dangers of methylmercury exposure and the 2004 Food and Drug Administration/Environmental Protection Agency (FDA/EPA) Advisory on fish consumption. It analyzes the extent to which particular groups of people living in the United States are vulnerable to toxic food consumption and the extent to which the advisories put them at risk.

Design/methodology/approach – The authors conducted a review of the literature related to methylmercury. In addition, they used archival information from government documents. They also analyzed the fish advisories and educational information posted on websites of each state's Women, Infants, and Children (WIC) program.

Findings – Methylmercury is a potent neurotoxin that can seriously affect fetal brain growth and development. Although the federal government promotes canned tuna as an inexpensive, beneficial protein source, recent reports indicate that a large proportion of America's favorite fish contains unsafe levels of methylmercury. Populations at risk for overconsumption include minorities and low-income groups, particularly recipients of federal subsidies such as WIC.

Practical implications – The FDA uses a “nuanced” message to offer consumers information about both the risks and benefits of eating fish. However, the advisory is not widely distributed and information on mercury levels in fish is not available at the point of sale. Minority populations are less likely to be aware of fish advisories and to change consumption habits. Thus, the target population remains largely uninformed and possibly misled about the risks and benefits of eating canned tuna.

Social implications – Based on new data, the federal government should recognize mercury as a hazard, update its consumption guidelines, and better monitor the mercury content in canned tuna. Outreach and educational programs should target minority and at-risk groups. The authors recommend an alternative precautionary approach in dealing with the health threats posed by methylmercury in tuna fish.

Children experience trauma more often than many early childhood educators realize. As many as 26% of children experience multiple trauma events such as abuse, neglect, parental substance abuse, parental incarceration, and so forth. Trauma impacts brain development in many negative ways that may have serious consequences on the child’s ability to learn, grow socially and emotionally, and develop physically. These brain changes also change how the child will play in the early childhood classroom, and information is given to help recognize the signs of trauma in children. The early childhood educator can make trauma-sensitive modifications in the classroom to assist the traumatized child’s ability to play out the problem. School counselors can be a resource for assisting early childhood teachers when working with traumatized children. A brief description of the importance of play therapy as a developmentally appropriate method to help traumatized young children is provided.

In this chapter, I review recent evidence on the developmental origins of health inequality. I discuss the origins of the education-health gradient, the long-term costs caused by early life adversity, and how early life experiences affect the biology of the body. Additionally, I provide complementary evidence on enrichment interventions which can at least partially compensate for these gaps. I highlight emerging lines of scientific inquiry which are likely to have a significant impact on the field. I argue that, while the evidence that early life conditions have long-term effects is now uncontroversial, the literature needs to be expanded both in a theoretical and empirical direction. On the one hand, a model linking early life origins to ageing needs to be developed; on the other hand, a better understanding of the mechanisms – both biological and socioeconomic – is required, in order to design more effective interventions.

The study of birth defects has increased in importance in recent years because the rate of infant mortality due to other causes (such as infection and nutritional disease) has decreased more quickly than has the rate of deaths due to birth defects. Today, abnormalities are detected in approximately 3 percent of newborn humans, and twice as many prenatally acquired defects are found in children after infancy as are discovered at birth. In addition, many of the more than 500,000 miscarriages and stillbirths that occur each year in the United States are due to abnormal fetal development.